Outline
DOI: 10.1093/bbb/zbag157 ISSN: 1347-6947
Fibroblast growth factor 21 promotes re-epithelialization through the ERK pathways during wound healing
Takumi Yamane, Ryosuke Konno, Manami Kimura, Okubo Mizuki, Yoshiaki Nakayama, Morichika Konishi, Yuichi OishiAbstract
We examined the effects of fibroblast growth factor (FGF)21 on re-epithelialization during wound healing. Wound closure and re-epithelialization were significantly delayed in FGF21 KO mice. For the in vitro assay, the FGF21-induced keratinocyte migration was reversed by extracellular signal-regulated kinase (ERK) inhibitors. These results suggest that FGF21 promotes re-epithelialization via the ERK pathways.
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