DOI: 10.4103/mjbl.mjbl_1618_23 ISSN: 1812-156X

Coronavirus Disease Infection Severity Correlation with Interleukin-22 Polymorphism among Iraqi Patients

Hussein Ali Noori, Layla Fouad Ali

Abstract

Background:

Numerous researched genes’ single nucleotide polymorphisms (SNPs) have been linked to the coronavirus disease (COVID-19) severity.

Objectives:

This study was conducted to determine whether the COVID-19 severity is correlated with the polymorphisms of two SNPs (rs1416125928 and rs2227485) in the interleukin (IL)-22 gene and, IL-22 level. Investigations were also conducted on how these polymorphisms affected IL-22 levels.

Materials and Methods:

For the detection of SARS-CoV-2, reverse-transcriptase polymerase chain reaction (RT-PCR) was used. Genomic DNA was isolated from blood samples for genotyping and ELISA tests to measure IL-22 levels. Polymerase chain reaction was used for genotyping.

Results:

The findings showed that patients’ levels of IL-22 considerably rise when compared to the control group of healthy individuals ( P < 0.01). And there is significant relationship in moderate group between rs1416125928 and IL-22 level ( P < 0.01) and non-significant relationship with severe and critical group both ( P ≤ 0.05). In rs2227485 there is no-significant relationship with IL-22 level in different types of severity ( P ≤ 0.05) in all of them.

Conclusion:

The study described here also demonstrated that IL-22 SNPs can alter IL-22 serum levels. The findings indicated that rs1416125928 and rs2227485 were active polymorphisms with no potential to influence IL-22 production and that there was no link between SNPs and blood levels of IL-22. Additionally, research was required to elucidate the molecular mechanisms that connected SNPs with IL-22 expression, the emergence of acute respiratory distress syndrome susceptibility, and prognosis. Our conclusions are limited by the sample size.

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