Ameliorating effect of α-glycosyl isoquercitrin treatment from gestation on disruption of hippocampal neurogenesis induced by maternal exposure to imidacloprid in rats

Maternal imidacloprid exposure impairs hippocampal neurogenesis via immunosuppression, oxidative stress, and BDNF-TrkB suppression. AGIQ effectively reverses these deficits by enhancing antioxidant capacity and AChE activity.